fructose
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Introduction
In nutrition: Found in high-fructose corn syrup
Mechanism of action
- intake correlates inversely with LDL particle size
- does not stimulate release of insulin or leptin
- may promote weight gain by not effectively inducing satiety[2]
- ingestion of fructose results in differences in blood flow (relative to glucose) in brain areas associated with hunger & reward (hypothalamus, thalamus, insula, cingulate cortex, fusiform gyrus, & striatum)[2]
- only sugar that raises serum uric acid
More general terms
More specific terms
Additional terms
Component of
- fructose/glucose/phosphoric acid
- fructose/glucose
- citrate/fructose/glucose
- FODMAP (Fermentable, Oligo-, Di-, Monosaccharides, & Polyols)
References
- ↑ Johnson RJ et al, Potential role sugar (fructose) in the epidemic of hypertension, obesity and the metabolic syndrome, diabetes, and cardiovascular disease. Am J Clin Nutr 2007, 86:899 PMID: https://www.ncbi.nlm.nih.gov/pubmed/17921363
Aeberli I et al, Fructose intake is a predictor of LDL particle size in overweight schoolchildren. Am J Clin Nutr 2007, 86:1174 PMID: https://www.ncbi.nlm.nih.gov/pubmed/17921399
Bray GA How bad is fructose? Am J Clin Nutr 2007, 86:895 PMID: https://www.ncbi.nlm.nih.gov/pubmed/17921361 - ↑ 2.0 2.1 2.2 Page KA et al. Effects of fructose vs glucose on regional cerebral blood flow in brain regions involved with appetite and reward pathways. JAMA 2013 Jan 2; 309:63. PMID: https://www.ncbi.nlm.nih.gov/pubmed/23280226
Database
- Kegg: http://www.genome.jp/dbget-bin/www_bget?
- PubChem: http://pubchem.ncbi.nlm.nih.gov/summary/summary.cgi?cid=5984
- PubChem: http://pubchem.ncbi.nlm.nih.gov/summary/summary.cgi?cid=439709
- PubChem: http://pubchem.ncbi.nlm.nih.gov/summary/summary.cgi?cid=3426
- PubChem: http://pubchem.ncbi.nlm.nih.gov/summary/summary.cgi?cid=716