coal workers' pneumoconiosis
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Epidemiology
coal workers
Pathology
- begins as anthracosis
- coal macules
- < 4 mm in size
- consists of macrophages, fibroblasts, reticulin & collagen fibers
- collections of coal macules around small airways cause bronchiolar dilatation & focal spongy emphysema
- no increased risk of malignancy or tuberculosis
History
Clinical manifestations
- non-specific bronchitis may occur with inhalation of large amounts of coal dust
- melanoptysis (expectoration of black sputum)
Diagnostic procedures
- pulmonary function testing:
- generally normal unless the patient smokes
- slight decrease in FEV1 may be related to centrilobular emphysema
- distinguishes obstructive vs restrictive pattern if abnormal
Radiology
- chest X-ray
- tiny nodular infiltrates in the upper lung zones
- micronodules (< 7 mm in diameter)
- macronodules (> 7 mm in diameter)
Management
- remove from exposure
More general terms
More specific terms
References
- ↑ Mayo Internal Medicine Board Review, 1998-99, Prakash UBS (ed) Lippincott-Raven, Philadelphia, 1998, pg 759
- ↑ Medical Knowledge Self Assessment Program (MKSAP) 16, American College of Physicians, Philadelphia 2012
- ↑ Kuempel ED, Wheeler MW, Smith RJ Contributions of dust exposure and cigarette smoking to emphysema severity in coal miners in the United States. Am J Respir Crit Care Med. 2009 Aug 1;180(3):257-64 PMID: https://www.ncbi.nlm.nih.gov/pubmed/19423717