hypoactive delirium
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Etiology
- uncontrolled pain
- pharmaceuticals
Epidemiology
- postoperative common, especially after nerve block wears off
- accounts for ~50% of delirium cases
Clinical manifestations
- features of delirium & depression
Laboratory
- plasma ammonia if cirrhosis & suspected hepatic encephalopathy
- sensitivity 47%, specificity 78%
- of secondary importance in patients with cirrhosis to medication review
Diagnostic procedures
- electroencephalography if suspected non-convulsive status epilepticus*
* after other etiologies ruled out
Radiology
- CT of head of low yield (13%)[2]
Differential diagnosis
- depression vs hypoactive delirium [1]
- depression must be present for at least 2 weeks
- non-convulsive status epilepticus
Management
- treatment pain
- non-pharmacologic manamgement of delirium
- antipsychotics not indicated in the absence of agitated delirium with patient presenting risk to themselves or others
More general terms
References
- ↑ 1.0 1.1 Geriatric Review Syllabus, 10th edition (GRS10) Harper GM, Lyons WL, Potter JF (eds) American Geriatrics Society, 2019
Geriatric Review Syllabus, 11th edition (GRS11) Harper GM, Lyons WL, Potter JF (eds) American Geriatrics Society, 2022 - ↑ 2.0 2.1 Akhtar H et al. Diagnostic yield of CT head in delirium and altered mental status- A systematic review and meta-analysis. J Am Geriatr Soc 2023 Mar; 71:946. PMID: https://www.ncbi.nlm.nih.gov/pubmed/36434820 https://agsjournals.onlinelibrary.wiley.com/doi/10.1111/jgs.18134