low cardiac output syndrome
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Etiology
- occurs most commonly in patients after cardiac surgery
Pathology
- transient decrease in systemic perfusion secondary to myocardial dysfunction
- imbalance between oxygen delivery & oxygen consumption resulting in metabolic acidosis
Management
- optimize myocardial contractility/ventricular function
- no benefit for inotrope levosimendan[2][3]
- improve diastolic dysfunction
- maintain adequate preload
- reduce afterload
- improve oxygen supply & demand
- allow time for the ventricle to recover
More general terms
References
- ↑ Masse L, Antonacci M. Low cardiac output syndrome: identification and management. Crit Care Nurs Clin North Am. 2005 Dec;17(4):375-83, x PMID: https://www.ncbi.nlm.nih.gov/pubmed/16344207
- ↑ 2.0 2.1 Mehta RH, Leimberger JD, van Diepen S et al. Levosimendan in patients with left ventricular dysfunction undergoing cardiac surgery. N Engl J Med 2017 Mar 19 <PubMed> PMID: https://www.ncbi.nlm.nih.gov/pubmed/28316276 Free Article <Internet> http://www.nejm.org/doi/10.1056/NEJMoa1616218
- ↑ 3.0 3.1 Landoni G, Lomivorotov VV, Alvaro G et al. Levosimendan for hemodynamic support after cardiac surgery. N Engl J Med 2017 Mar 21 <PubMed> PMID: https://www.ncbi.nlm.nih.gov/pubmed/28320259 Free Article <Internet> http://www.nejm.org/doi/10.1056/NEJMoa1616325